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Correspondence |

Salt-Wasting and Hyponatremia in Neuroleptic Malignant Syndrome

Ronald J. Gurrera, MD
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Copyright 2007 American Medical Association. All Rights Reserved. Applicable FARS/DFARS Restrictions Apply to Government Use.

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Arch Neurol. 2007;64(7):1058-1058. doi:10.1001/archneur.64.7.1058-a
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With their report1 of cerebral salt-wasting syndrome in a patient with neuroleptic malignant syndrome (NMS), Dr Lenhard and colleagues have taken a potentially important step toward explicating the pathophysiology of NMS. Previous work2 suggested that salt-wasting caused by profound diaphoresis might explain electrolyte and fluid imbalances in NMS and the frequent co-occurrence of severe diaphoresis, hyponatremia, and polydipsia in that disorder. However, Lenhard et al propose that elevated levels of adrenomedullary brain natriuretic peptide (BNP) caused the severe hyponatremia observed in their patient. This alternative mechanism is intriguing because it represents a more direct consequence of the dysregulated hyperautonomic state that is central to NMS.

Although BNP is synthesized and secreted primarily by heart tissue, particularly in response to volume overload,3 comprehensive clinical evaluation excluded relevant cardiac pathology in their patient.1 Within the porcine nervous system, BNP concentrations are highest in spinal cord, followed by medulla-pons and striatum; only modest levels are found in the hypothalamus.4 This pattern is even more pronounced in the rat central nervous system, where initial studies5 6 found significant BNP levels only in spinal cord, and subsequent work7 demonstrated BNP-staining cell bodies that were more widely distributed but dispersed in clusters throughout sympathetic preganglionic cell columns. These data imply a functional linkage between sympathetic nervous system activation and BNP levels and support the hypothesis that elevated BNP associated with sympathoadrenal hyperactivity may cause hyponatremia in NMS. Obviously, extreme diaphoresis may also contribute to salt-wasting in some cases of NMS.

AUTHOR INFORMATION

Correspondence: Dr Gurrera, Psychiatry Service, VA Boston Healthcare System and Department of Psychiatry, Harvard Medical School, 940 Belmont St (116A), Brockton, MA 02301 (ronald.gurrera@va.gov).

Financial Disclosure: None reported.

REFERENCES

Lenhard  T, Kulkens  S, Schwab  S. Cerebral salt-wasting syndrome in a patient with neuroleptic malignant syndrome. Arch Neurol 2007;64 (1) 122- 125
PubMed
Gurrera  RJ. Diaphoresis and dehydration during neuroleptic malignant syndrome: preliminary findings. Psychiatry Res 1996;64 (2) 137- 145
PubMed
Azzazy  HME, Christenson  RH. B-type natriuretic peptide: physiologic role and assay characteristics. Heart Fail Rev 2003;8 (4) 315- 320
PubMed
Ueda  S, Minamino  N, Sudoh  T, Kangawa  K, Matsuo  H. Regional distribution of immunoreactive brain natriuretic peptide in porcine brain and spinal cord. Biochem Biophys Res Commun 1988;155 (2) 733- 739
PubMed
Aburaya  M, Suzukil  E, Minamin  N, Kangawal  K, Tanaka  K, Matsu  H. Concentration and molecular forms of brain natriuretic peptide in rat plasma and spinal cord. Biochem Biophys Res Commun 1991;177 (1) 40- 47
PubMed
Ogawa  Y, Nakao  K, Mukoyama  M.  et al.  Rat brain natriuretic peptide: tissue distribution and molecular form. Endocrinology 1990;126 (4) 2225- 2227
PubMed
Saper  CB, Hurley  KM, Moga  MM.  et al.  Brain natriuretic peptides: differential localization of a new family of neuropeptides. Neurosci Lett 1989;9629- 34
PubMed

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Lenhard  T, Kulkens  S, Schwab  S. Cerebral salt-wasting syndrome in a patient with neuroleptic malignant syndrome. Arch Neurol 2007;64 (1) 122- 125
PubMed
Gurrera  RJ. Diaphoresis and dehydration during neuroleptic malignant syndrome: preliminary findings. Psychiatry Res 1996;64 (2) 137- 145
PubMed
Azzazy  HME, Christenson  RH. B-type natriuretic peptide: physiologic role and assay characteristics. Heart Fail Rev 2003;8 (4) 315- 320
PubMed
Ueda  S, Minamino  N, Sudoh  T, Kangawa  K, Matsuo  H. Regional distribution of immunoreactive brain natriuretic peptide in porcine brain and spinal cord. Biochem Biophys Res Commun 1988;155 (2) 733- 739
PubMed
Aburaya  M, Suzukil  E, Minamin  N, Kangawal  K, Tanaka  K, Matsu  H. Concentration and molecular forms of brain natriuretic peptide in rat plasma and spinal cord. Biochem Biophys Res Commun 1991;177 (1) 40- 47
PubMed
Ogawa  Y, Nakao  K, Mukoyama  M.  et al.  Rat brain natriuretic peptide: tissue distribution and molecular form. Endocrinology 1990;126 (4) 2225- 2227
PubMed
Saper  CB, Hurley  KM, Moga  MM.  et al.  Brain natriuretic peptides: differential localization of a new family of neuropeptides. Neurosci Lett 1989;9629- 34
PubMed

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