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Comment & Response |

Computer-aided Therapeutics in Treating Autoimmune Encephalitis

Jagannadha Avasarala, MD, PhD1
[+] Author Affiliations
1Division of Neurology, Neuroscience Associates, Department of Medicine, Greenville Health System, Greenville, South Carolina
JAMA Neurol. 2016;73(1):127-128. doi:10.1001/jamaneurol.2015.3516.
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To the Editor I read with great interest the article in JAMA Neurology byJoubert and colleagues1 that detailed the clinical features of autoimmune encephalitis (AE) mediated by α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor antibodies. In this regard, the father of modern-day AE research, Josep Dalmau, and his colleagues2 have published numerous articles that define this category of diseases. Of these disorders, the best characterized, perhaps, is the anti–N-methyl-D-aspartate receptor encephalitis receptor (NMDAR) variant, which is the prototypical AE syndrome. Despite these rapid advances, treatment options for patients with AE remain rather mundane and target-nonspecific. Unfortunately, neurology as a science is driven by discovery rather than therapy—this gap is probably narrowing, if slowly.

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January 1, 2016
Bastien Joubert, MD; Pierre Paoletti, PhD; Jérôme Honnorat, MD, PhD
1University Lyon 1, University Lyon, Lyon, France2Hospices Civils de Lyon, Neuro-oncologie, Hôpital Neurologique, Bron, France
3Institut de Biologie de l’Ecole Normale Supérieure (IBENS), Ecole Normale Supérieure, CNRS UMR 8197, INSERM U1024, Paris, France
1University Lyon 1, University Lyon, Lyon, France2Hospices Civils de Lyon, Neuro-oncologie, Hôpital Neurologique, Bron, France4Centre National de Référence pour les Syndromes Neurologiques Paranéoplasiques, Hospices Civils de Lyon, Hôpital Neurologique, Bron, France5Lyon Neuroscience Research Center, Neuro-Oncology and Neuro-Inflammation Team, INSERM, UMR-S1028, CNRS, UMR-5292, Lyon, France
JAMA Neurol. 2016;73(1):128. doi:10.1001/jamaneurol.2015.3519.
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